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STUDY OF INHERITENCE IN SEGERATING POPULATIONS OF WHEAT FOR YIELD RELATED TRAITS
Abstract
The research was initiated by developing five generations (P1, P2, F1, F2 and F3) of the seven cross combinations of wheat including RK-23 × Maxipak, RK-23 × Inqilab, RK-23 × Seher, RK-23 × Zincol, RK-23 × Shahkar, RK-23 × Borlague and RK-23 × Gold. The five generations of each cross were sown at two locations i.e Agricultural research institute (ARI), D. I. Khan and Cereal crops research institute (CCRI), PirSabak, Nowshehra. Data were collected on yield parameters including grains spike-1, grain weight spike-1, 1000 grain weight and yield plant-1. Analysis of variance (ANOVA) shows that effect of environment was significant for all the traits in all cross combinations Significant/highly significant differences among P1, P2, F1, F2 and F3 generations were calculated for grain weight spike-1 in all cross combination, for grains spike-1 in all cross combinations excluding RK-23× Inqilab, for 1000 grains spike-1 in all cross combinations excluding RK-23×Seher, RK-23 × Zincol and RK-23×Borlague and for yield plant-1 in all cross combinations excluding RK-23 × Gold. Significant generations × location interaction was noted in most of the cross combinations for all the traits. The cross combination RK-23 × Shahkar showed highest mean for grain weight spike-1 (3.72 gm ) and grains spike-1 (90 grains) in F1 generation. The highest mean (47 gm) for 1000 grain weight and yield plant-1 (63 gm) was recorded in F1 generation of RK-23 × Seher and RK-23 × Inqilab, respectively. Gene action analysis shows that grain weight spike-1, 1000 grain weight and grains spike-1 were controlled by dominant besides additive and additive types of gene action in most of the cross combinations, however some cross combinations also showed dominant × dominant epistasis. Inheritence of yield plant-1 was controlled by additive and epistasis types of gene effects in most of the cross combinations, however few cross combinations were also under the influence of dominant gene effect.
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